G1 Therapeutics, Inc. Reports Earnings Results for the Second Quarter and Six Months Ended June 30, 2023
August 02, 2023 at 04:00 pm IST
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G1 Therapeutics, Inc. reported earnings results for the second quarter and six months ended June 30, 2023. For the second quarter, the company reported revenue was USD 42.39 million compared to USD 10.57 million a year ago. Net income was USD 8.71 million compared to net loss of USD 39.45 million a year ago. Basic earnings per share from continuing operations was USD 0.17 compared to basic loss per share from continuing operations of USD 0.92 a year ago. Diluted earnings per share from continuing operations was USD 0.14 compared to diluted loss per share from continuing operations of USD 0.92 a year ago.
For the six months, revenue was USD 55.34 million compared to USD 17.48 million a year ago. Net loss was USD 18.89 million compared to USD 88.64 million a year ago. Basic loss per share from continuing operations was USD 0.37 compared to USD 2.08 a year ago. Diluted loss per share from continuing operations was USD 0.37 compared to USD 2.08 a year ago.
G1 Therapeutics, Inc. is a commercial-stage biopharmaceutical company. The Company is focused on the development and commercialization of small molecule therapeutics for the treatment of patients with cancer. The Company's lead commercial product, COSELA (trilaciclib), is a therapy indicated to proactively help protect bone marrow (myeloprotection) from the damage of chemotherapy. Its product portfolio consists of Trilaciclib and Lerociclib, both of which are CDK4/6 inhibitors, and a Cyclin-dependent kinase 2 (CDK2) inhibitor. Trilaciclib is a novel therapy designed to transiently arrest cells that are dependent on CDK4/6 for proliferation, including hematopoietic stem and progenitor cells (HSPCs), in the G1 phase. Lerociclib is a differentiated clinical-stage oral CDK4/6 inhibitor being developed for use in combination with other targeted therapies in multiple oncology indications. COSELA is a short-acting intravenous CDK4/6 inhibitor. Its CDK2 is an internally discovered inhibitor.